Marty Martin, Psy.D.

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The Centers for Disease Control and Prevention now says it’s investigating more than 450 cases of severe lung illnesses believed to be linked to vaping. CBS News’ Dean Reynolds has the story of one teen who barely survived.

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Featuring panelists Sarah E. Kucharski, Mark Freeman, Danielle Edges, and Allison Ferlito.

Mental health and chronic disease management are often interwoven topics in science. But what about the human side of the story? Whether or not individuals meet the diagnostic criteria for mental diagnoses, we all long for optimal mental and physical health to attain good quality of life.

How do we put a personal face to the feelings that hypoglycemia, or chronic pain, or other physical health struggles invoke in terms of our emotions? What moments have changed the way we think about mental health and chronic disease, and how can we encourage these same poignant healthcare interactions in the lives of others?

This panel explores just how vital this conversation is in chronic disease and mental health management by featuring multifaceted patient, caregiver, and mental health advocate perspectives. In order to facilitate this conversation for all patients, we must raise the topic from the very source: ePatients themselves. Therefore, this panel is ePatient-centric in its selection of presenters. We will identify crucial learning moments in understanding our own mental health, while promoting a transparent discussion that remains much-needed in humanizing healthcare.

Ah-Ha! moments relevant to the following topics will be discussed:
– Why the words we use matter so much
– Barriers to care
– Continuum of “before, during, and after” mental health becomes a focus of care management; early intervention in normalizing this conversation
– Caregiver resources and how to talk to children/teens about difficult health topics
– Managing multiple diagnoses
– Curtailing advocacy “burnout”
– The panel would also be open to answering audience questions, time-permitting.
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Read the article: http://dx.doi.org/10.1038/s41598-018-34393-9

Garg et al. “Evaluating polymicrobial immune responses in patients suffering from tick-borne diseases.” Scientific Reports (2018). doi: 10.1038/s41598-018-34393-9

Video produced by https://www.researchsquare.com

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(USMLE topics) Pathophysiology of HDN, Signs and Symptoms, Prevention and Treatment options.
This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/all-animations/heart-and-blood-circulation-videos/-/medias/3c52a09d-6812-4cf2-a9c4-98041f83f74d-hemolytic-disease-of-the-newborn-narrated-animation
©Alila Medical Media. All rights reserved.
Voice by Ashley Fleming

Support us on Patreon and gain early access to our videos and FREE images downloads: patreon.com/AlilaMedicalMedia

All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Hemolytic disease of the newborn, HDN, is a condition in which red blood cells of a newborn infant, or a perinatal fetus, are destroyed prematurely, resulting in anemia. HDN occurs when the blood types of the mother and baby are incompatible. A blood type refers to the presence or absence of a certain antigen, on the surface of a person’s red blood cells. Incompatibility happens when the baby has an antigen that the mother does not have. The mother’s immune system interprets the antigen as “foreign” and produces antibodies to target the cells carrying it for destruction.
While in principle HDN may occur with mismatch in any blood group, severe cases most commonly involve D-antigen of the Rh system. Specifically, HDN may develop if an Rh-negative mother, having no D-antigen, carries an Rh-positive fetus, with D-antigen. The first mismatch pregnancy, however, is usually not at risk. This is because the placenta normally does a good job separating the mother’s blood from the fetal blood, preventing the fetal red blood cells from being exposed to the mother’s immune system. However, at birth, or if a miscarriage or abortion occurs, the tearing of the placenta exposes fetal blood to the mother, who then responds by producing anti-D antibodies. Because antibody production takes some time, it does not affect the first baby; but if the mother is again pregnant with another Rh-positive fetus, her antibodies, being small enough to cross the placenta, can now cause hemolysis.
The first mismatch pregnancy may be at risk if the mother has previously been exposed to the antigen in other ways, such as through blood transfusion or sharing needles, or if the placental barrier is breached because of trauma, or medical procedures early in the pregnancy.
Anemia can cause heart failure, respiratory distress, and edema. Infants born with HDN also develop jaundice due to the accumulation of bilirubin, a yellow product of hemoglobin breakdown. Because red blood cells are destroyed rapidly and infants are unable to excrete bilirubin effectively, its levels rise quickly within 24h of birth. Bilirubin is toxic for brain tissues and may cause irreversible brain damage in a condition known as kernicterus. Other signs of HDN include enlarged liver, spleen, and presence of immature red blood cells, erythroblasts, in the blood. Some of these signs can be detected before birth, with ultrasound imaging.
HDN that involves D-antigen can now be effectively prevented with anti-D antibody. It is given to Rh-negative mothers during and soon after the first mismatch pregnancy. The antibody binds to fetal blood cells that leak into the mother’s blood, either destroying them, or hiding them from the mother’s immune system, thus preempting the mother’s immune response.
Infants born with HDN are usually treated with intravenous fluid, and phototherapy, a procedure in which a certain spectrum of light is used to convert bilirubin to a form that is easier for the infant to excrete.
Severe anemia may be treated with:
– blood transfusion,
– intravenous immunoglobulin G therapy, which works by blocking the destruction of antibody-coated red blood cells.
– and exchange transfusion, where the baby’s blood is essentially replaced with Rh-negative donor blood. This procedure is very effective at removing bilirubin and reducing the destructive effect of the mother’s antibody, but may have adverse effects.
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(USMLE topics, cardiology) Life and death of erythrocytes, anemia and polycythemia. This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/all-animations/heart-and-blood-circulation-videos/-/medias/5519909f-97f9-44f6-a806-0c87af9addb7-red-blood-cell-disorders-narrated-animation
©Alila Medical Media. All rights reserved.
Voice by Ashley Fleming
Support us on Patreon and gain early access to our videos and FREE images downloads: patreon.com/AlilaMedicalMedia
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Production of red blood cells occurs in the red bone marrow, and is stimulated by erythropoietin, EPO. EPO is secreted predominantly by the kidneys. The kidneys sense oxygen levels in the blood and adjust EPO secretion accordingly to the body’s needs.
Red cells live about 100 to 120 days. With age, the cells lose their elasticity. Without protein synthesis, they are unable to repair themselves. Worn-out red cells are detected in the spleen, which serves as a quality control center. The spleen has a network of very narrow channels which test the agility of erythrocytes. Healthy cells can bend and fold to squeeze through, while old cells, being rigid and fragile, get stuck and are destroyed by macrophages. Parts of the dead cells are salvaged to make new cells. Part of the heme is secreted into bile and disposed in feces.
The number of red blood cells is strictly regulated and has important clinical significance. Common measurements include red blood cell count, hematocrit, and hemoglobin concentration.
An imbalance between the rate of red cell production and death can result in their deficiency, known as anemia, or excess, known as polycythemia.
Anemia can be caused by blood loss, insufficient erythrocyte production, or their premature destruction.
Insufficient red cell production can result from:
+ deficiency of any of the nutrients that are required for their formation,
+ impaired kidney function, which leads to lower secretion of EPO,
+ or destruction of the bone marrow tissue responsible for red cell production. This can happen because of inherited mutations, autoimmune diseases, or exposure to chemicals, drugs or radiation; but causes are unknown for many cases. Reduced erythropoiesis is known as hypoplastic anemia, while complete cessation of red cell production is called aplastic anemia.
Inappropriate destruction of red blood cells, also called hemolytic anemia, can be inherited or acquired. The inherited forms are usually due to defects within red cells themselves, such as abnormalities in hemoglobin structure, while acquired hemolytic anemia can be caused by toxins, drugs, autoimmune diseases, infection, overactive spleen, or blood group mismatch.
Anemia results in low oxygen levels in the blood, known as hypoxemia. Mild anemia causes weakness and confusion, while severe anemia may lead to organ failure due to lack of oxygen and is life-threatening.
Excess red cell production, or polycythemia, can be primary or secondary. Primary polycythemia, or polycythemia vera, is a form of blood cancer, where the bone marrow produces too many blood cells. Secondary polycythemia, on the other hand, is a consequence of low oxygen state, which induces the kidneys to produce more erythropoietin, subsequently leading to more erythrocytes. Causes include smoking, air pollution, emphysema, living at high altitudes, and physical strenuous conditioning in athletes.
Excess red cells may increase blood volume, blood pressure, and viscosity. This augments the risks for blood clot formation, which may lead to heart attacks, strokes, and pulmonary embolism. The heart also has to work harder to manage larger amount of thicker blood and heart failures may result.

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How is periodontal disease treated? Periodontitis and its influence on health.

This 3D video from Clínica Médico Dental Pardiñas (http://j.mp/cPardinas) explains the periodontal disease (periodontitis) and its relationship with the rest of the human body. It is shown how tartar accumulates on the teeth and how bacteria present in it can spread throughout the bloodstream. This is especially important in patients with heart problems, or that have received a valve replacement or transplantation. These bacteria can reach the heart valves and adhere to them, causing an infection called endocarditis.

Surgical treatment with different instruments and the resolution of the disease is also shown.

Periodontitis results in the destruction of the bone holding the tooth in place, causing all of the teeth to be lost if it is not treated in time. It appears as inflamed gums with presence of periodontal pockets (that form beneath the gum), which can be detected using a small probe that measures the distance between the gum and the bone, the mobility or exposure of the root of the tooth. Periodontitis is very rare in children and adolescents (1 case in every 10,000). However, its frequency increases with age (10% between 30-40 years old; 25%-30% between 50-60 years old).

Symptoms include: gums separated from the teeth, bleeding, bad breath, tooth mobility, receding gums, etc. When bone resorbs, a space between the gums and bone starts to create, this is called periodontal pocket. Here bacteria accumulate, proliferate and the patient can no longer sanitize properly.
To address this problem, it is necessary to eliminate the calculus, both superficial and deeper, and reshape the gum to remove the periodontal pocket, leaving the gum at the bone level. For this purpose, various instruments such as curettes and ultrasounds are used.
The objective is to remove all plaque and damaged tissue, leaving the tooth free of plaque and tartar.
After this procedure, the gums may retract, and the roots of the teeth may be exposed, which can cause sensitivity and aesthetic problems. What is achieved with this procedure is not regain the lost bone, but slow the progression of the disease, getting a state of health of the gums and eliminating periodontal pockets, so that the patient can have a good oral hygiene.

What is the cause of periodontal disease (periodontitis)?
It is caused by bacteria that accumulate in tartar and dental plaque, which sticks around the teeth and that if we do not eliminate, they get inserted between the tooth and the gum. These bacteria create toxins that damage and develop an infectious inflammatory process, which involves the destruction of the tissues that support the teeth. There must also be a predisposing individual (genetic) factor that is key, and that is what explains that patients with very little tartar can develop an aggressive periodontal disease, and vice versa, patients with a lot of tartar hardly have bone losses in the jaws.

#periodontitis #periodontaldisease #periodontist

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Facet joint, facet syndrome, facet disease- what does it all mean?!?

And most importantly, how can you find relief and live your best life?

In this video, Dr. Jeff Langmaid explains how your facet joints act as the “knuckles” of your spine and allow to you move, bend, and twist.

Also, we look at what happens when your facets become arthritic, and how you can on the road to relief.
____________________________________________

Jeff Langmaid is a chiropractor, author, and speaker. He built the worlds largest research-focused chiropractic communication service, The Evidence Based Chiropractor. He also is the founder of Chiro Office Coverage. Throughout his career he has been featured on Yahoo Health, MSN Health, CBS, ABC, Prevention Magazine, Chiropractic Economics, Circle of Docs, The Chiropractic Philanthropist, amongst others.

The Evidence Based Chiropractor YouTube channel is designed to provide immediate, action-able knowledge regarding chiropractic, communication, business, and marketing. All to help you grow and develop your practice.

Learn more at-

The Evidence Based Chiropractor: http://www.TheEvidenceBasedChiropractor.com
Chiro Office Coverage: http://www.ChiroOfficeCoverage.com
Facebook: http://www.facebook.com/TheEvidenceBasedChiropractor

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Gum disease – aka “periodontal disease” aka “periodontitis”– is the most common reason behind adult tooth loss. The number one reason people lose their teeth is not because of cavities – its because of gum disease. Let’s talk about how to treat gum disease!

Gum disease is preventable, but only if you catch it in the earliest stage of gingivitis. Once more aggressive periodontal disease kicks in, you’ll require professional treatment to prevent tooth loss.

The severity of your gum disease can put your health at risk. As medical issues and periodontitis go hand in hand, it’s crucial to treat them together instead of as two separate issues.

Treating gum disease starts at home with good daily brushing and flossing, followed up with professional cleanings with your hygienist.

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▶GINGIVITIS VS PERIODONTITIS VIDEO:

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▶DEEP CLEANING (SRP) VIDEO:

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Dr. Mark Bonner has developed a “Cure” for gum disease that does not involve traditional surgical procedures.

CLICK BELOW to learn more about the procedure

Antimicrobial Gum Disease Therapy

We can determine whether a patient is infected with pathogenic micro-organisms using a microscope. By identifying the specific bacteria present in the mouth, Dr. Joseph Nemeth can eliminate them and help prevent the disease from reoccurring.

If you have any questions or would like to schedule an appointment, please visit our website,

Home

We are happy to help you.

Joseph R. Nemeth DDS & Associates
29829 Telegraph Road #111
Southfield, MI 48034
(248) 357-3100

We DO NOT accept Medicare or Medicaid at our office.
If you are a new patient, we require that a deposit be made prior to your appointment in order to reserve your time.

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Instagram: https://www.instagram.com/drnemethdds/ The information contained in the multimedia content (“Video Content”) has been made available for informational and educational purposes only. Joseph R. Nemeth, DDS & Associates does not make any representation or warranties with respect to the accuracy, applicability, fitness, or completeness of the Video Content. Joseph R. Nemeth, DDS & Associates does not warrant the performance, effectiveness or applicability of any sites listed or linked to in any Video Content. The Video Content is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or other qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read or seen on the Site. Joseph R. Nemeth, DDS & Associates hereby disclaims any and all liability to any party for any direct, indirect, implied, punitive, special, incidental or other consequential damages arising directly or indirectly from any use of the Video Content, which is provided as is, and without warranties.

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Hiii to all the nursing aspirants there!!! Through this lecture I had explained various levels of disease prevention through valid examples. After listening to this lecture the students will be able to understand the concept of disease prevention easily.

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